Baking soda is an effective aid in the treatment of arthritis. It is an autoimmune disease. The body is suffering pain signals in joints. In the given condition, the body trying to find out ways of managing pain. One method is to release painkilling chemicals, called endorphins. It is released in happiness. Another method to take treatment by a doctor or by using a home remedy.
Baking soda, also known as sodium bicarbonate has been used as a home remedy due to its antacid properties. Its benefits work greatly in sorting out arthritis pain. It became popular also to use as a home remedy in treating various health issues. For example, half a teaspoon is often taken to relieve acid reflux or heartburn, or it can be used to whiten teeth.
New research had been done at the Medical College of Georgia at Augusta University that baking soda works against autoimmune disease such as Rheumatoid Arthritis. Researchers performed tests of baking soda first on experimental animals and then humans.
Baking soda gives a signal to special types of cells called Mesothelial Cells, preventing the body from the harmful autoimmune response and give the response to the body is safe and not under attack. Mesothelial cells provide a lining to the internal organs and prevent sticking one cell from another. Now the action of baking soda starts if intake it. Baking soda encourages the stomach to secrete more gastric juice or acid which is able to digest food quicker and mesothelial cells work greatly inlining other cells and permit no interference of spleen army of macrophages or white blood cells cleared. Certainly drinking bicarbonate affects the spleen and we think it’s through the mesothelial cells. Mesothelial cells communicate with the organs they line using small projections called microvilli, and the medium through which they send their message is the neurotransmitter acetylcholine.
The study authors note that those who drank the baking soda experienced a solution of the shift in the kind of immune cells activated in the spleen. In fact, pro-inflammatory macrophages (M1) dwindled in number, while the levels of anti-inflammatory cells (M2) rose.